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Abstract Oxidative stress and TNF are critically involved in the initiation and progression of non-alcoholic fatty liver disease (NAFLD)

Neuronal Glud1 (glutamate dehydrogenase 1) over-expressing mice: increased glutamate formation and synaptic release, loss of synaptic activity, and adaptive changes in genomic expression

One such option would be utilizing antioxidant therapies to neutralize reaction oxygen species (ROS) in the retina that underlie RP progression