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Hepatic PGC-1 overexpression exacerbates APAP-induced hepatotoxicity and mortality in mice Since Pgc-1 is one of the master regulators of liver metabolism and given its increased expression observed when mice were challenged with HFD, we decide to decipher whether PGC-1 induction could contribute to the worsening of APAP-induced hepatotoxicity
That's roughly 50 times less than the research doses
2011 in the Journal of Applied Physiology characterized BPC-157's effects on tendon fibroblast outgrowth through increased FAK phosphorylation and downstream paxillin activation, with measurable effects on cell migration rate and tendon-derived fibroblast survival after mechanical injury
