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It targets the disc extracellular matrix directly and may help maintain structural integrity before significant herniation occurs
The mechanisms of TB-500 are still being investigated, but known actions of TB4 include: Binding as an actin-binding protein to inhibit the polymerization of globular actin (G-actin) into filamentous actin (F-actin), known as actin sequestration, which results in higher G-actin levels and increased cellular motility [23, 24] TB4s ability to increase cellular motility may help progenitor cells to reach sites of injury and initiate tissue recovery [25]
The meta-analysis of these studies found that omega-3 supplementation reduced the risk of cardiovascular disease in patients with diabetes by 7% (Huang 2023)
JAMA psychiatry 77 (12), 12461255
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