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The downregulation of ChaC1, along with other genes, appears to contribute to the mechanisms by which lung adenocarcinoma cells develop resistance to cisplatin, highlighting the UCA1-TXNIP axis as a potential therapeutic target for overcoming drug resistance (Zhou et al., 2020)
It is, however, available without prescription as a nutritional supplement
These results also suggest that a modification of the concentration of NADP+/NADPH is not the cause of the increased activity of the PPP enzymes
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