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As prolonged expression of matrix metalloproteases and other degrading enzymes are present in the eye following cataract surgery (322), while new basement membrane protein synthesis is disrupted, it would be expected that the lens capsule would change properties at extended times PCS, which could compromise IOL-lens capsule interactions, allowing the escape of cells onto the posterior capsule where they have the potential to contribute to light scatter and late onset PCO (134, 322)

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