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Mutants of the candidate residues were subsequently compared with wild-type ScGrx7 in steady-state kinetic measurements using HEDS and GSSCys as a non-glutathione and glutathione disulfide substrate, respectively
Distinct types of cell death and the implication in diabetic cardiomyopathy
The resistance mechanism is squalene epoxidase (ERG1) mutations (F397L and related variants)
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