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We run the GSH program and the APAP program together - they share the substrate GSH, which is drawn down by the overdose and replenished by the GSH synthesis pathway
This pathway mainly regulates ferroptosis by inducing and activating the expression of SREBP1, which can target and activate the downstream SCD1, and SCD1 can inhibit cell ferroptosis by increasing the content of MUFA
All data were expressed as meanstandard deviation (s.d.)
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