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This is especially true if you have diabetes, cardiovascular conditions, thyroid disorders, or kidney disease, all of which can be affected by changes in GLP-1 receptor agonist therapy
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Interestingly, GCGR-deficient mice are resistant to streptozotocin (STZ)-induced beta cell destruction and hyperglycaemia [111], which led to the suggestion that the presence of glucagon might be more important for the hyperglycaemia associated with type 1 diabetes than the lack of insulin [13]
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