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The findings revealed that irregular activation of HGF/c-MET enhanced TGF- release from cancer cells, reduced CTL penetration at the tumor site, and eventually caused immune evasion

This is sometimes done when the initial solution is too concentrated for accurate dosing

(2003) suggested that the cleft may enable large peptide substrates to access the active site, whereas the narrower tunnel formed by the -propeller could serve as a conduit for exit of dipeptide cleavage products