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tBid induces alterations of mitochondrial fatty acid oxidation flux by malonyl-CoA-independent inhibition of carnitine palmitoyltransferase-1
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Difference in muscle metabolism caused by metabolism disorder of rainbow trout liver exposed to ammonia stress
In some hypertrophic and failing contexts, inadequate FAO contributes to energetic failure, and pan-ERR agonists that enhance cardiac fatty acid metabolism and mitochondrial function improved heart failure phenotypes ( 3.2 Glucose uptake, glycolysis, pyruvate-lactate flux, and anaplerosis Increased glucose uptake and glycolysis are among the most reproducible features of hypertrophic remodeling, but the fate of glucose carbon varies
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