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Large-scale, population-based cohort studies have demonstrated an association between T2D and an increased risk of developing PD, with hazard ratios ranging from 1.2 to 1.5, depending on the study design and population [6, 25,26,27,28]
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Additionally, the co-localization or fusion of lysosomal CTSB with trypsinogen in ZGs appears to be a prerequisite for AP development
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