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Although the main factor in ferroptosis prevention is believed to be GPX4, other crucial players include the GPX4-independent ferroptosis suppressor protein 1 (FSP1) [38]

It simultaneously activates three key metabolic receptors: GLP-1 (glucagon-like peptide-1), GIP (glucose-dependent insulinotropic polypeptide) and the receptor at glucagon This triple synergistic action makes it one of the most studied peptides in research on obesity and metabolic disorders