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Apolipoprotein C-III deficiency accelerates triglyceride hydrolysis by lipoprotein lipase in wild-type and apoE knockout mice

This convergence across all three mouse models indicates that interferon-responsive and antigen-presentation programs in the OL lineage are not unique to autoimmune demyelination but instead represent a broader injury-adaptive response

Adult male dopamine -hydroxylase knockout (DBH / ) mice and controls (DBH +/+ or DBH +/ ) at 9- to 12-months of age were treated with ICV PBS (DBH WT Control, N = 3), no surgery (DBH KO Controls, N = 5), or 10 ng/hr leptin (both DBH WT, N = 5 and DBH KO, N = 2)

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