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These seem to be the mechanisms by which ROS participate in the induction of low-grade chronic inflammation, cellular dysfunction, and implicitly in the promotion and aggravation of chronic conditions (e.g., cardiovascular, pulmonary, renal, metabolic, neurological, malignancies) ( Regarding kidney damage, OS is correlated in the literature with both antioxidant depletion and excessive ROS production, serving as both a cause and an effect of renal degradation
Pancreatic mucinous cystic neoplasms following GLP-1 receptor agonists use: a report of two cases with literature review
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