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This positioning places DSIP within sleep architecture modulation research rather than acute sleep induction mechanisms
10.1177/1534734620907773 27 ChandrasekaranK.ChoiJ.ArvasM
in fact, these treatments promoted UPR mt and mitophagy, restoring mitochondrial and organismal function, and even stimulated the reduction of amyloid deposits in these models 114
Low SAM may limit methyl-donor reserve and could interfere with SAM-dependent creatine synthesis
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