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Together, these peptides act synergistically to accelerate healing, reduce inflammation, and strengthen tissue resilience 1-5

Although rodent models provide important tools for AD research, it is difficult for these models to fully simulate the complex pathophysiological processes of human AD because of the significant differences in brain structure, metabolic characteristics, and immune responses between humans and experimental animals (Qian et al., 2024)

Without rigorous testing and process controls, a compounded drug could be sub-potent (containing too little API to be effective) or super-potent (containing a dangerously high dose)

From the co-localization analysis, we infer that increased nausea or vomiting is associated with greater BMI loss efficacy (Extended Data Fig