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Evidence from multiple fields, including metabolism, cell biology, and clinical endocrinology, collectively suggests that within the uterine fibroid microenvironment, E 2 and progesterone may induce excessive ROS accumulation through three pathways: enzyme metabolic imbalance, mitochondrial dysfunction, and disruption of the antioxidant system
Powerful Antioxidant: Protects skin cells from free radical damage caused by UV exposure and environmental stressors
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