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To determine whether specific acidic and intrinsically disordered regions of CSQ directly contribute to its solubility-enhancing effect, we generated domain-deletion variants of CSQ-anti-VEGF-scFv

Furthermore, Sun et al., showed that HuSC-Exos protect against kidney ischemia-reperfusion injury by reducing ferroptosis via lncRNA taurine upregulated gene 1, which regulates ACSL4 through serine arginine-rich splicing factor 1 interaction, suggesting a potential acute kidney injury therapy [72, 106] (Table 1)

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