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One is that GLP-1 RAs cause rapid drops in blood glucose levels, potentially creating a hypoxic environment in the retina, which is an ideal setting for abnormal angiogenesis to take hold

GLP-1RA activates the GLP-1 receptor in platelets, through an cyclic adenosine monophosphate/protein kinase (AMP/PKA)-dependent mechanism to prevent systemic inflammation, vascular dysfunction, and end-organ damage, thereby significantly reducing the microvascular thrombosis induced by endotoxin blood disease and mortality (Steven et al., 2016) (Tests on human vascular endothelial cells exposed to LPS have demonstrated that GLP-1 analogs reduce vascular permeability and lower the levels of pro-inflammatory cytokines (Wonhwa et al., 2016)

Tesofensine: This peptide works in the brain to help suppress appetite while also increasing the bodys ability to burn fat, a process known as fat oxidation

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