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Increased VEGF expression and neovascularization in irradiated wounds Improved wound closure rates compared to control in compromised tissue Demonstrates efficacy even in radiation-impaired healing environments Comprehensive review of GHK and GHK-Cu: stimulation of collagen, elastin, and connective tissue accumulation

doi: 10.3389/fimmu.2022.833355 262 SinhaR.SinhaI.CalcagnottoA.TrushinN.HaleyJ

Its main appeal is that it supports the biology of repair

Experimental vascular studies indicate that BPC-157 modulates endothelial nitric oxide synthase activity by altering the interaction between endothelial nitric oxide synthase and its regulatory binding partner caveolin-1 [1][2]