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On the contrary, activation of the TSS pathway under physiological cystine supplement is achieved under DMF treatment through enhanced CBS expression and cystine uptake to protect against ferroptosis in SCD erythroblasts
C3G not only attenuates cellular and tissue oxidative stress but also reduces Fe 2+ levels, inhibits nuclear receptor coactivator 4 (NCOA4) expression, and concurrently regulates transferrin receptor protein 1 (TfR1) downregulation and ferritin heavy chain 1 (FTH1) upregulation
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