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In striking contrast, HS at physiological concentrations exhibits unique protective mechanisms: it upregulates the expression of tight junction proteins (zonula occludens-1, Claudin-1) to repair the physical barrier, and promotes goblet cell differentiation as well as mucin glycosylation (Bi et al., 2023)
While genetic approaches have been valuable for identifying bacterial genes required for GSH import and metabolism, tools for directly assessing bacterial utilization of host GSH remain limited
H., Yu, K
K., Tracey, K., Yang, H., LeRoith, D., Brownstein, M
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