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Carnitine in Schizophrenia Carnitine has potential diagnostic and therapeutic effects on schizophrenia (67, 68)
Research suggests GHK-CU has a relatively short half-life, making twice-daily administration theoretically superior for sustained gene expression effects
Specifically, the researchers suggested that the cleavage of caspase-3 and its substrate called Poly ADP-ribose polymerase (PARP) appeared markedly reduced, by ~43% and ~77% respectively, suggesting that the peptide complex may suppress this cascade of programmed cell death
This damage can alter the permeability and fluidity of cell membranes, ultimately leading to changes in cellular structure and function, and may trigger cell death [57, 58]
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