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In Crohns Disease, the immune response is predominantly mediated by Th1 cells (producing IFN- and TNF-) and Th17 cells (secreting IL-17 and IL-22)
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Mitochondrial and lysosomal dysfunction, dysregulated iron metabolism, and accumulation of ROS are considered to be pivotal elements in the pathogenesis of PD, ultimately leading to neuronal degeneration and death (Matak et al., 2016
It performs an array of critical functions, including detoxifying drugs, protecting macromolecules from oxidative damage, and maintaining immune functions [ 1]
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