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When NRF2 translocates to the nucleus, it binds antioxidant response elements (AREs) in the promoter regions of GST genes, upregulating their expression

J Am Soc Nephrol (2004) 15(8):213951

Future work will use longitudinal profiling and single-cell sequencing to assess cell-type-specific GSH roles 5 Conclusion In conclusion, by leveraging multi-omics integration, the present study identified GSH metabolism as a pivotal node in SAE, while providing a comprehensive molecular framework for SAE pathogenesis, and linking microglia-driven neuroinflammation to hippocampal oxidative damage