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When the body is in a state of excess oxidative stress the expression of this enzyme is induced
One possible hypothesis leading to this observation could be related to the increasing concentration of TG in the remaining VLDL and rising of TG antiport with HDL-C at the same time.[] One study showed a contrasting result by reporting the effect of carnitine on the HDL-C level
Despite being mechanistically different, pyroptosis, ferroptosis, apoptosis, necroptosis act synergistically via shared signaling nodes in DKD, with mitochondrial ROS (mtROS) and serving as a pivotal common mediator (30, 112, 176, 207)
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