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TRIM37 and TRIM28 act with AP2 to remodel chromatin and repress somatic gene programs To further explore the molecular mechanisms underlying the repression of gene expression by the TRIM37TRIM28 complex, we first explored the relationship between chromatin accessibility and gene transcription following Trim37 inactivation using RNA-seq and ATAC-seq analyzes of PGCs at E9.25 (Supplementary information, Fig
Cluster headaches rare but extremely severe headaches, often centered around one eye
Thrombus formation and the subsequent activation of the coagulation cascade with final generation of fibrin is facilitated by the mCRP-mediated enhancement of tissue factor on the endothelial cell surface, platelet aggregation and thrombus growth (207, 208) (Figure 7)
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