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Those ligand-target bindings would include benzodiazepine, opioid, (1)- and (2)-adrenergic, serotonergic and cholinergic receptors 85 and, notably, the fascinating issue of the anti-nociceptive role of melatonin seems to be mediated by MT1 and MT2 receptors themselves 86 , 87,88

Rather than replacing individual peptides, these combinations bring together complementary mechanisms associated with vascular biology, cellular migration, extracellular matrix remodeling, and connective tissue physiology

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