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The copper interaction anomalies in these proteins contribute to misfolding and aggregation that is linked to neurodegeneration
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Accordingly, AhR activation with various canonical ligands (ITE (5 M), TCDD (10 nM), BaP (5 M), FICZ (5 M), and Indirubin (5 M)) in SKR cells (24 h) induced significantly ACSL4 and PLIN3 protein expression (Appendix Fig S3A)
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