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Complement degradation products such as C3a and C5a released by complement system activation can also indirectly enhance ferroptosis sensitivity by promoting iron ion efflux and lipid peroxidation, further expanding the regulatory dimension of inflammation on the cell death network
Beyond directly influencing pathological changes in the AD brain, BAs may provide neuroprotective benefits by modulating the microbiotagutbrain (MGB) axis, which supports memory function
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