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Systemic application of 3-methyladenine markedly inhibited atherosclerotic lesion in ApoE/ mice by modulating autophagy, foam cell formation and immune-negative molecules
Specifically, exenatide treatment over 48 to 60 weeks increased tyrosine phosphorylation of insulin receptor substrate 1 (IRS-1) in neuronal-derived extracellular vesicles (exosomes), alongside activation of downstream signaling proteins such as total Akt and phosphorylated mTOR [61]
