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In this regard, we have successively documented that the formation of a Bcl-2/p38MAPK heterocomplex into the cytoplasm is the genuine responsible for the inhibition of vRNPs nuclear export

Hst3p, a key sirtuin histone deacetylase, mediates H3K56 deacetylation to maintain chromatin repression, and its inhibition leads to genome-wide H3K56ac enrichment, which further dysregulates the transcription of virulence-associated genes including adhesins, phenotypic switching regulators and hyphal formation-related factors, thereby disrupting yeast-hyphae transition, white-opaque switching and host adhesion processes essential for C

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[32] To maintain a balance of intracellular superoxide, organisms have developed strategies to protect against overexposure