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The reaction is catalyzed by a family of enzymes called Glutathione S-transferases (GSTs), which facilitate a nucleophilic attack, covalently binding the sulfur atom of glutathione directly to the toxic Phase I metabolite
Understanding these metabolic effects has implications for research into diabetes and related metabolic conditions
The immune system needs time to recognize the medication as foreign, produce antibodies, and mount a localized response upon subsequent exposures
A missense variant in mitochondrial Amidoxime reducing component 1 gene and protection against liver disease
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