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By modulating downstream genes like ACSL1 and RSAD2, the drug prevents the lipid accumulation required for M2 macrophage survival
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An analysis of 103 G6PD mutants causing G6PD deficiency in humans, as against 52 G6PD sequences from 45 different organisms, has shown that most mutations (74%) are in highly and moderately conserved (50%99% of similarity) AAs, whereas few mutations are in fully conserved or in poorly conserved AAs ( Fig
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