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Further analyses of residual tumours indicated that Fento-1 induced oxidation of membrane lipids and the production of lysophospholipids, which affected preferentially CD44 high over CD44 low cancer-cell subpopulations (Fig

All findings are from short-duration preclinical models only

Survodutide adds the glucagon-receptor axis on top of GLP-1 signaling, and in preclinical models this addition drove greater weight loss than a maximally effective semaglutide dose attributed to the extra energy-expenditure component

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