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Where estrogen works to restore the hormonal environment that governs fat distribution and metabolic regulation, GLP-1 therapy directly reduces caloric intake by recalibrating appetite signaling, improves glycemic control through multiple mechanisms, and drives the reduction of visceral fat mass that estrogen alone cannot reliably produce in the setting of significant metabolic burden
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L-carnitine buffers excess acetyl-CoA inside the mitochondria by forming acetylcarnitine, which can reduce metabolic stress, support carbohydrate oxidation and improve efficiency during higher exercise intensities
