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GLP-1 receptor activation complements this by promoting mitochondrial turnover, the process by which damaged or dysfunctional mitochondria are cleared and replaced, and by reducing the production of reactive oxygen species, the unstable molecules that accumulate during metabolic stress and accelerate cellular damage
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10.3390/ijms23179655 Int
While sommarutide is undoubtedly an exciting potential treatment option, there is still a lack of clear clinical data on the mechanism of action of GLP-1 agonists in NASH, such as whether GLP-1 agonists have a direct, independent role in improving NASH, or simply by improving body weight, insulin resistance, and so on
