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Previous research has shown that excessive nitric oxide (NO) production, increased levels of oxidative stress, and cerebrovascular hypoperfusion are mayor pathomechanisms responsible for AD (Lepara et al
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SP, AB, GJ, and AD corrected the manuscript
This suggests that FOXO4-DRI may selectively target and eliminate senescent cells, potentially alleviating some dysfunctions associated with aged Leydig cells, such as reduced testosterone synthesis
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