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Die Arbeiten von Pickart und Mitarbeitern (2012, 2014) zeigten, dass GHK-Cu das Genexpressionsprofil von einem gealterten zu einem jngeren Zustand umlenkt
10.1080/02757540.2022.2126461 3 AlbahriG.AlyamaniA
Patients with MDD exhibit KP dysregulation, often marked by an overproduction of quinolinic acid, an N-Methyl-D-aspartic acid receptor (NMDAR) agonist that drives excitotoxicity, alongside reduced production of kynurenic acid, an NMDAR antagonist that protects from excitotoxicity and has anti-inflammatory effects
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