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Studies in humans and animals with non-alcoholic fatty liver disease show that CD44 plays a key role in the inflammation and liver damage in NAFLD.[ref] Other genetic studies have looked at genes in pathways known to be involved in acetaminophen metabolism, including genes related to sulfation, glucuronidation, and glutathione production

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For TJA, priority endpoints include wound complications, prolonged drainage, hematoma requiring intervention, early readmission, and PJI, with follow-up sufficient to capture early revision risk

The viral S-protein serves as an anchor to interact with host tissue, followed by sequential cleavage of S-protein to facilitate viral entry 57,58