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High homocysteine could also mean you have an MTHFR gene mutation, which makes you less efficient at converting inactive folate to the active form of folate that breaks down homocysteine

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As illustrated in Scheme 1B, once the GCMMR NPs were endocytosed by cancer cells through the RGD-mediated targeting affinity, the outer PEG was destroyed in the acidic TME, following which MNSs converted GSH into glutathione disulfide (GSSG) and released MET to alleviate the hypoxia of TME as well as interfere with energy supply by inhibiting mitochondria-mediated respiration

For anyone considering GHK-Cu, my advice is simple: give it time, stay consistent, and keep your expectations realistic