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For example, UV-B-induced flavonol accumulation can be suppressed during immune activation through antagonistic MYB regulation, redirecting metabolic resources toward defense-associated phenolics and antimicrobial compounds (Logemann and Hahlbrock, 2002
Ecotox 18:385402 Hahn ME, Hestermann EV (2008) Receptor-mediated mechanisms of toxicity
This C-terminal fragment is used in research to investigate mechanisms of lipid metabolism and adipocyte biology, including experimentally reported effects on lipolysis and related pathways, without the full spectrum of growth-promoting activity associated with the intact hGH molecule
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