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Headache represents another occasionally reported effect, potentially related to the peptide's vasodilatory properties

By activating transcription factors like NF-B and activator protein 1 (AP-1) and upregulating kinases like phosphatidylinositol 3-kinase (PI3K) and mitogen-activated protein kinases (MAPKs), ROS generation can cause pro-inflammatory responses (Chen and Kunsch 2004)

It may prevent lipid peroxidation [5], can regulate T-cell blockage [30], and its effects on NF-B inhibit matrix metalloproteinase 9 (MMP) [37, 38]

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