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In global terms, hypothalamic pathways involved in autophagy, neurotransmitter release, cytoskeletal remodeling (RHO GTPases signaling), vesicle-mediated transport and intracellular signaling (PI3K/AKT activation) were upregulated in PWA mice treated with GLP1/E, while pathways related with vesicle transport, oxidative stress, DNA repair, metabolism and immune system were downregulated by GLP1/E treatment (Fig
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Although causality cannot be demonstrated, researchers characterized the findings as compelling and suggest they support the development of a prospective randomized trial to determine whether GLP-1 receptor agonists offer disease-modifying potential as a component of sickle cell disease therapy
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