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Increased microvascular permeability brought on by ROS causes inflammatory cells to migrate and exacerbates the inflammatory process ( SalB protects endothelial cells from oxidative stress injury by up-regulating the expression of the tight junction gene, reducing the permeability of injured endothelial cells, and counteracting the increase in ROS caused by BLM through the MAPK and NF-B signaling pathways ( SalB exhibits multiple features of anti-oxidant, inflammation inhibition, and immune modulation, which are indispensable for the treatment of PF ( In summary, one of S

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Clinical trials published in 2026 continue to validate this assertion

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