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Similar findings were also reported in PMA-stimulated mouse and human neutrophils where inhibition of G6PD significantly impaired their ability to undergo oxidative burst (64)
These properties highlight its potential as a therapeutic agent for managing inflammation and improving recovery in various conditions
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The activation of GLP-1R can reduce the size of cerebral infarction by enhancing cell survival signaling pathways, reducing ischemia-reperfusion injury, promoting brain repair, and inhibiting inflammatory response and oxidative stress (8588)
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