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In long-term evolution, human UA kinase factors and a series of promoter gene mutations have caused humans to have higher UA levels than other mammals (Figure 1) (Shuang Liang et al., 2018)
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[21] Individuals of 677TT are predisposed to mild hyperhomocysteinemia (high blood homocysteine levels), because they have less active MTHFR available to produce 5-methyltetrahydrofolate (which is used to decrease homocysteine)
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