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The peptide has been characterized in tendon, ligament, muscle, gut, vascular, neural and bone injury models, where it consistently accelerates structural recovery, restores barrier function and rescues vascular perfusion through a coordinated upregulation of growth factor receptors, the nitric oxide system and angiogenic signaling [13]

Beyond GLP-1/GIP dual agonism, triple-hormone receptor agonists such as retatrutide further expand this framework by engaging GIP, GLP-1, and glucagon receptors within a single molecule (109)

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