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Furthermore, we provide first data that the GSH level in human neurons may be mediated by NMDAR activation since the protective effect of enhanced network activity could be blocked with an NMDAR antagonist APV, consistent with the previous report (Fig
Inhibition of endocytosis with dynasore blocked both VEGFR2 internalization and the pro-angiogenic effects of BPC 157, confirming the essential role of this process
[The activity of erythrocyte and platelet glutathione reductase and glutathione-S-transferase in paranoid schizophrenia]
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