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Ex/Em = 488/576 nm)) [481]

Some of these molecules have also entered clinical trials and we will possibly welcome in the future the approval of a GST inhibitor or pro-drug for the treatment of patients

This is particularly relevant in the cancer context, where the tumor microenvironment often promotes oxidative stress and altered cellular signaling pathways, contributing to the rapid turnover of RBCs

Preclinical and Phase II clinical trials (e.g., in idiopathic pulmonary fibrosis) demonstrate TD139s potent antiplatelet effects, reducing arterial thrombosis and myocardial ischemia/reperfusion (MI/R) injury without increasing the risk of bleeding [57, 58] (Fig